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Kidney TransplantationArchives

Robotic‐assisted native pyeloureterostomy with indocyanine green, after kidney transplantation

 Published on 24/07/2024 |  Original article (Full-text)  | Nour Khalil et al. | Asian Journal of Endoscopic Surgery 2024; 17(3): 13329

Kidney transplantation (KT) is the optimal treatment for end‐stage renal disease, offering improved quality of life and increased life expectancy and quality of life. Nevertheless, KT is a complex procedure associated with postoperative complications that can reduce the intended benefits. Bladder...

A personalised delisting strategy enables successful kidney transplantation in highly sensitised patients with preformed donor‐specific anti HLA antibodies

 Published on 17/07/2024 |  Original article (Full-text)  | Sandra García‐Jiménez et al. | HLA: Immune Response Genetics 2024; 103(6): 15572

The single‐antigen bead (SAB) Luminex assay has greatly improved DSA detection in kidney transplantation (KT), and the established association between preDSA and early acute antibody‐mediated rejection (aAMR) risk and long‐term graft loss, has directed transplant teams towards preDSA...

Risk factors for Pneumocystis jirovecii pneumonia after kidney transplantation: A systematic review and meta‐analysis

 Published on 10/07/2024 |  Original article (Full-text)  | Bingjie Cheng et al. | Clinical Transplantation 2024; 38(5): 15320

Since the first kidney transplant (KT), the field of transplantation has been transformed. KT has become the most effective kidney replacement treatment for chronic kidney disease, which can extend life expectancy and enhance the quality of life. Despite the current significant improvement in survival...

Oxalate Nephropathy After Kidney Transplantation: Risk Factors and Outcomes of Two Phenotypes

 Published on 03/07/2024 |  Original article (Full-text)  | Neetika Garg et al. | Clinical Transplantation 2024; 38(6): 15368

Oxalate or oxalic acid is a dicarboxylic acid that is derived either from diet or from endogenous metabolism of ascorbic acid and some amino acids. There is no human enzyme that degrades oxalate further, and normally it is freely filtered and excreted by the kidneys [1]. Primary hyperoxaluria represents...